uri,href,identifier,attrs.Abstract,attrs.Author,attrs.DOI,attrs.Date,attrs.ISSN,attrs.Issue,attrs.Journal,attrs.Keywords,attrs.Language,attrs.Notes,attrs.Pages,attrs.Title,attrs.Volume,attrs.Year,attrs.\.reference_type,attrs._record_number,attrs._uuid,attrs.reftype,child_publication
/reference/3a561ebc-1125-4ce9-8292-854a3b46b37a,https://data.globalchange.gov/reference/3a561ebc-1125-4ce9-8292-854a3b46b37a,3a561ebc-1125-4ce9-8292-854a3b46b37a,"The degree to which aeroallergens are contributing to the global increase in pediatric allergic disease is incompletely understood. We review the evidence that links climate change to changes in aeroallergens such as pollen and outdoor mold concentrations and, subsequently, aeroallergen association with pediatric allergic disease. We specifically explore the evidence on both the exacerbation and the development of allergic disease in children related to outdoor pollen and mold concentrations. Pediatric allergic diseases include atopic dermatitis or eczema, allergic rhinitis or hay fever, and some types of asthma in children, typically defined as < 18 years of age. We discuss how the timing of aeroallergen exposure both in utero and in childhood could be associated with allergies. We conclude that the magnitude and type of health impacts due to climate change will depend on improved understanding of the relationship between climatic variables, multiple allergen factors, and allergic disease. Improved public-health strategies such as adequate humidity control, optimum air filtration and ventilation, and improved anticipatory public-health messaging will be critical to adaptation.","Sheffield, P. E.; Weinberger, K. R.; Kinney, P. L.",10.1002/msj.20232,Jan-Feb,0027-2507,1,"Mount Sinai Journal of Medicine","Air Pollutants/*adverse effects; Allergens/*adverse effects; Child; *Climate Change; Humans; Hypersensitivity/*etiology",eng,"1931-7581 Sheffield, Perry E Weinberger, Kate R Kinney, Patrick L 5T32 HD049311/HD/NICHD NIH HHS/United States ES09089/ES/NIEHS NIH HHS/United States L40 ES017745-01/ES/NIEHS NIH HHS/United States P30 ES009089/ES/NIEHS NIH HHS/United States P30 ES009089-09/ES/NIEHS NIH HHS/United States T32 ES007322-09/ES/NIEHS NIH HHS/United States T32 HD049311-03/HD/NICHD NIH HHS/United States T32ES007322/ES/NIEHS NIH HHS/United States Journal Article Research Support, N.I.H., Extramural Review United States Mt Sinai J Med. 2011 Jan-Feb;78(1):78-84. doi: 10.1002/msj.20232.",78-84,"Climate change, aeroallergens, and pediatric allergic disease",78,2011,0,18552,3a561ebc-1125-4ce9-8292-854a3b46b37a,"Journal Article",/article/10.1002/msj.20232
/reference/3a569dc6-4596-488a-8c30-c0b6f884677f,https://data.globalchange.gov/reference/3a569dc6-4596-488a-8c30-c0b6f884677f,3a569dc6-4596-488a-8c30-c0b6f884677f,,"Kleinpeter, M. A.",10.2215/cjn.08690811,,1555-905X,10,"Clinical Journal of the American Society of Nephrology",,,,2337-2339,"Disaster preparedness for dialysis patients",6,2011,0,17781,3a569dc6-4596-488a-8c30-c0b6f884677f,"Journal Article",/article/10.2215/cjn.08690811
/reference/3ac8bd8a-540b-43c5-9881-6470785b3e87,https://data.globalchange.gov/reference/3ac8bd8a-540b-43c5-9881-6470785b3e87,3ac8bd8a-540b-43c5-9881-6470785b3e87,"BACKGROUND: Few epidemiologic studies have investigated associations of air pollution with cognition in older adults, and none has specifically compared associations across particle sources. We investigated whether exposure to particulate air pollution, characterized by size and source, was associated with cognitive function and decline in cognitive function. METHODS: We included participants of the Whitehall II cohort who were residents of greater London and who attended the medical examination in study wave 2007-2009 (n = 2867). Annual average concentrations of particulate matter (PM) (PM10 and PM2.5 from all sources and from traffic exhaust) were modeled at resolution of 20 x 20 m for 2003-2009. We investigated the relationship between exposure to particles and a cognitive battery composed of tests of reasoning, memory, and phonemic and semantic fluency. We also investigated exposure in relation to decline in these tests over 5 years. RESULTS: Mean age of participants was 66 (standard deviation = 6) years. All particle metrics were associated with lower scores in reasoning and memory measured in the 2007-2009 wave but not with lower verbal fluency. Higher PM2.5 of 1.1 mug/m (lag 4) was associated with a 0.03 (95% confidence interval = -0.06 to 0.002) 5-year decline in standardized memory score and a 0.04 (-0.07 to -0.01) decline when restricted to participants remaining in London between study waves. CONCLUSIONS: This study provides support for an association between particulate air pollution and some measures of cognitive function, as well as decline over time in cognition; however, it does not support the hypothesis that traffic-related particles are more strongly associated with cognitive function than particles from all sources.","Tonne, C.; Elbaz, A.; Beevers, S.; Singh-Manoux, A.",10.1097/ede.0000000000000144,Sep,1531-5487,5,Epidemiology,,eng,"1531-5487 Tonne, Cathryn Elbaz, Alexis Beevers, Sean Singh-Manoux, Archana MR/K013351/1/Medical Research Council/United Kingdom R01 AG013196/AG/NIA NIH HHS/United States R01 AG034454/AG/NIA NIH HHS/United States R01AG 013196/AG/NIA NIH HHS/United States R01AG 034454/AG/NIA NIH HHS/United States Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't United States Epidemiology. 2014 Sep;25(5):674-81. doi: 10.1097/EDE.0000000000000144.",674-681,"Traffic-related air pollution in relation to cognitive function in older adults",25,2014,0,18194,3ac8bd8a-540b-43c5-9881-6470785b3e87,"Journal Article",/article/10.1097/ede.0000000000000144
/reference/3aebbe26-1f02-42e8-84bd-100532a82484,https://data.globalchange.gov/reference/3aebbe26-1f02-42e8-84bd-100532a82484,3aebbe26-1f02-42e8-84bd-100532a82484,,"Rotkin-Ellman, M.; Solomon, Gina; Gonzales, Christopher R.; Agwaramgbo, Lovell; Mielke, Howard W.",10.1016/j.envres.2009.09.004,,0013-9351,1,"Environmental Research",,,,19-25,"Arsenic contamination in New Orleans soil: Temporal changes associated with flooding",110,2010,0,19316,3aebbe26-1f02-42e8-84bd-100532a82484,"Journal Article",/article/10.1016/j.envres.2009.09.004
/reference/3b1803a8-da7d-4b78-8ea8-605336acf55b,https://data.globalchange.gov/reference/3b1803a8-da7d-4b78-8ea8-605336acf55b,3b1803a8-da7d-4b78-8ea8-605336acf55b,,"Charron, Dominique F.; Thomas, M.Kathleen; Waltner-Toews, D.; Aramini, Jeffery J.; Edge, Tom; Kent, Robert A.; Maarouf, A.R.; Wilson, Jeff",10.1080/15287390490492313,,1087-2620,20-22,"Journal of Toxicology and Environmental Health, Part A: Current Issues",,,,1667-1677,"Vulnerability of waterborne diseases to climate change in Canada: A review",67,2004,0,19185,3b1803a8-da7d-4b78-8ea8-605336acf55b,"Journal Article",/article/10.1080/15287390490492313
/reference/3b392444-e0b0-4298-9e3e-431e1dcd35fa,https://data.globalchange.gov/reference/3b392444-e0b0-4298-9e3e-431e1dcd35fa,3b392444-e0b0-4298-9e3e-431e1dcd35fa,,"Mares, Dennis",10.1007/s11524-013-9791-1,,1468-2869,4,"Journal of Urban Health",,,,768-783,"Climate change and levels of violence in socially disadvantaged neighborhood groups",90,2013,0,19215,3b392444-e0b0-4298-9e3e-431e1dcd35fa,"Journal Article",/article/10.1007/s11524-013-9791-1
/reference/3b3ef9ac-3b80-4a9a-9ce3-a7809e5b0862,https://data.globalchange.gov/reference/3b3ef9ac-3b80-4a9a-9ce3-a7809e5b0862,3b3ef9ac-3b80-4a9a-9ce3-a7809e5b0862,,"Pope, C. Arden, III; Turner, Michelle C.; Burnett, Richard T.; Jerrett, Michael; Gapstur, Susan M.; Diver, W.Ryan; Krewski, Daniel; Brook, Robert D.",10.1161/CIRCRESAHA.116.305060,,1524-4571,1,"Circulation Research",,,,108-115,"Relationships Between Fine Particulate Air Pollution, Cardiometabolic Disorders, and Cardiovascular Mortality",116,2015,0,19201,3b3ef9ac-3b80-4a9a-9ce3-a7809e5b0862,"Journal Article",/article/10.1161/CIRCRESAHA.116.305060
/reference/3baf471f-751f-4d68-9227-4197fdbb6e5d,https://data.globalchange.gov/reference/3baf471f-751f-4d68-9227-4197fdbb6e5d,3baf471f-751f-4d68-9227-4197fdbb6e5d,,"Walthall, C.; Backlund, P.; Hatfield, J.; Lengnick, L.; Marshall, E.; Walsh, M.; Adkins, S.; Aillery, M.; Ainsworth, E.A.; Amman, C.; Anderson, C.J.; Bartomeus, I.; Baumgard, L.H.; Booker, F.; Bradley, B.; Blumenthal, D.M.; Bunce, J.; Burkey, K.; Dabney, S.M.; Delgado, J.A.; Dukes, J.; Funk, A.; Garrett, K.; Glenn, M.; Grantz, D.A.; Goodrich, D.; Hu, S.; Izaurralde, R.C.; Jones, R.A.C.; Kim, S-H.; Leaky, A.D.B.; Lewers, K.; Mader, T.L.; McClung, A.; Morgan, J.; Muth, D.J.; Nearing, M.; Oosterhuis, D.M.; Ort, D.; Parmesan, C.; Pettigrew, W.T.; Polley, W.; Rader, R.; Rice, C.; Rivington, M.; Rosskopf, E.; Salas, W.A.; Sollenberger, L.E.; Srygley, R.; Stockle, C.; Takle, E.S.; Timlin, D.; White, J.W.; Winfree, R.; Wright-Morton, L.; Ziska, L.H.",,,,,,,,,186,"Climate Change and Agriculture in the United States: Effects and Adaptation",,2012,10,3329,3baf471f-751f-4d68-9227-4197fdbb6e5d,Report,/report/usda-techbul-1935
/reference/3bc7615b-d281-4fd6-a367-c94022d367a8,https://data.globalchange.gov/reference/3bc7615b-d281-4fd6-a367-c94022d367a8,3bc7615b-d281-4fd6-a367-c94022d367a8,,"Wadsworth, Martha E.; Santiago, Catherine DeCarlo; Einhorn, Lindsey",10.1080/10615800902855781,,1477-2205,4,"Anxiety, Stress & Coping",,,,413-432,"Coping with displacement from Hurricane Katrina: Predictors of one-year post-traumatic stress and depression symptom trajectories",22,2009,0,16384,3bc7615b-d281-4fd6-a367-c94022d367a8,"Journal Article",/article/10.1080/10615800902855781
/reference/3bd47363-8f13-4c90-b52d-26e7ff47f216,https://data.globalchange.gov/reference/3bd47363-8f13-4c90-b52d-26e7ff47f216,3bd47363-8f13-4c90-b52d-26e7ff47f216,"There is broad scientific consensus that Earth's climate is warming rapidly and at an accelerating rate. Human activities, primarily the burning of fossil fuels, are very likely ( > 90% probability) to be the main cause of this warming. Climate-sensitive changes in ecosystems are already being observed, and fundamental, potentially irreversible, ecological changes may occur in the coming decades. Conservative environmental estimates of the impact of climate changes that are already in process indicate that they will result in numerous health effects to children. The nature and extent of these changes will be greatly affected by actions taken or not taken now at the global level. Physicians have written on the projected effects of climate change on public health, but little has been written specifically on anticipated effects of climate change on children's health. Children represent a particularly vulnerable group that is likely to suffer disproportionately from both direct and indirect adverse health effects of climate change. Pediatric health care professionals should understand these threats, anticipate their effects on children's health, and participate as children's advocates for strong mitigation and adaptation strategies now. Any solutions that address climate change must be developed within the context of overall sustainability ( the use of resources by the current generation to meet current needs while ensuring that future generations will be able to meet their needs). Pediatric health care professionals can be leaders in a move away from a traditional focus on disease prevention to a broad, integrated focus on sustainability as synonymous with health. This policy statement is supported by a technical report that examines in some depth the nature of the problem of climate change, likely effects on children's health as a result of climate change, and the critical importance of responding promptly and aggressively to reduce activities that are contributing to this change.","Shannon, M. W.; Best, D.; Binns, H. J.; Forman, J. A.; Johnson, C. L.; Karr, C. J.; Kim, J. J.; Mazur, L. J.; Roberts, J. R.; Shea, K. M.",10.1542/peds.2007-2645,Nov,1098-4275,5,Pediatrics,"climate change; global warming; child; pediatric; health; sustainable; development",English,"Times Cited: 14 Shannon, Michael W. Best, Dana Binns, Helen J. Forman, Joel A. Johnson, Christine L. Karr, Catherine J. Kim, Janice J. Mazur, Lynnette J. Roberts, James R. Shea, Katherine M. Amer acad pediatrics Elk grove village","1149-1152 ","Global climate change and children's health",120,2007,0,4249,3bd47363-8f13-4c90-b52d-26e7ff47f216,"Journal Article",/article/10.1542/peds.2007-2645
/reference/3be13957-eae2-4796-8504-ef2597b91b09,https://data.globalchange.gov/reference/3be13957-eae2-4796-8504-ef2597b91b09,3be13957-eae2-4796-8504-ef2597b91b09,"Coastal storms can take a devastating toll on the public's health. Urban areas like New York City (NYC) may be particularly at risk, given their dense population, reliance on transportation, energy infrastructure that is vulnerable to flood damage, and high-rise residential housing, which may be hard-hit by power and utility outages. Climate change will exacerbate these risks in the coming decades. Sea levels are rising due to global warming, which will intensify storm surge. These projections make preparing for the health impacts of storms even more important. We conducted a broad review of the health impacts of US coastal storms to inform climate adaptation planning efforts, with a focus on outcomes relevant to NYC and urban coastal areas, and incorporated some lessons learned from recent experience with Superstorm Sandy. Based on the literature, indicators of health vulnerability were selected and mapped within NYC neighborhoods. Preparing for the broad range of anticipated effects of coastal storms and floods may help reduce the public health burden from these events. © 2013 Kathryn Lane et al.","Lane, K.; Charles-Guzman, K.; Wheeler, K.; Abid, Z.; Graber, N.; Matte, T.",10.1155/2013/913064,,1687-9813,,"Journal of Environmental and Public Health","cleaning; emergency shelter; flooding; hazard; health care delivery; health impact assessment; housing; human; hurricane; mental health; mould; outcomes research; priority journal; review; seashore; urban area; vulnerable population; water contamination",,"Cited By (since 1996):1 Export Date: 7 November 2013 Source: Scopus Art. No.: 913064 Language of Original Document: English Correspondence Address: Lane, K.; Bureau of Environmental Surveillance and Policy, New York City Department of Health and Mental Hygiene, New York, NY 10013, United States; email: klane1@health.nyc.gov Funding Details: 1UE1EH000757-01, CDC, Centers for Disease Control and Prevention",1-13,"Health effects of coastal storms and flooding in urban areas: A review and vulnerability assessment",2013,2013,0,4697,3be13957-eae2-4796-8504-ef2597b91b09,"Journal Article",/article/10.1155/2013/913064
/reference/3be13d86-b82e-41dd-8687-6db274d6823d,https://data.globalchange.gov/reference/3be13d86-b82e-41dd-8687-6db274d6823d,3be13d86-b82e-41dd-8687-6db274d6823d,,"Lefebvre, Kathi A.; Robertson, Alison",10.1016/j.toxicon.2009.05.034,,1879-3150,2,Toxicon,,,,218-230,"Domoic acid and human exposure risks: A review",56,2010,0,18356,3be13d86-b82e-41dd-8687-6db274d6823d,"Journal Article",/article/10.1016/j.toxicon.2009.05.034
/reference/3bfcb39e-f3ee-4d20-8f53-77c8487599b4,https://data.globalchange.gov/reference/3bfcb39e-f3ee-4d20-8f53-77c8487599b4,3bfcb39e-f3ee-4d20-8f53-77c8487599b4,,"Sapkota, A.
Symons, J.M.
Kleissl, J.
Wang, L.
Parlange, M.B.
Ondov, J.
Breysse, P.N.
Diette, G.B.
Eggleston, P.A.
Buckley, T.J.",10.1021/es035311z,,0013-936X,1,"Environmental Science & Technology",,,,24-32,"Impact of the 2002 Canadian forest fires on particulate matter air quality in Baltimore City",39,2005,0,2747,3bfcb39e-f3ee-4d20-8f53-77c8487599b4,"Journal Article",/article/10.1021/es035311z
/reference/3bfe667e-40cb-4dc3-9da4-75df2ab699ce,https://data.globalchange.gov/reference/3bfe667e-40cb-4dc3-9da4-75df2ab699ce,3bfe667e-40cb-4dc3-9da4-75df2ab699ce,,"Foulds, S.A.; Brewer, P.A.; Macklin, M.G.; Haresign, W.; Betson, R.E.; Rassner, S.M.E.",10.1016/j.scitotenv.2013.12.079,,1879-1026,,"Science of the Total Environment",,,,165-180,"Flood-related contamination in catchments affected by historical metal mining: An unexpected and emerging hazard of climate change",476-477,2014,0,19320,3bfe667e-40cb-4dc3-9da4-75df2ab699ce,"Journal Article",/article/10.1016/j.scitotenv.2013.12.079
/reference/3cfcb05a-9a2a-4fb1-89be-6377ea83e5d0,https://data.globalchange.gov/reference/3cfcb05a-9a2a-4fb1-89be-6377ea83e5d0,3cfcb05a-9a2a-4fb1-89be-6377ea83e5d0,,"Wagner, Carola; Adrian, Rita",10.4319/lo.2009.54.6_part_2.2460,,1939-5590,6_part_2,"Limnology and Oceanography",,,,2460-2468,"Cyanobacteria dominance: Quantifying the effects of climate change",54,2009,0,18951,3cfcb05a-9a2a-4fb1-89be-6377ea83e5d0,"Journal Article",/article/10.4319/lo.2009.54.6_part_2.2460
/reference/3d663a99-383d-4728-9336-ff558cd83ad1,https://data.globalchange.gov/reference/3d663a99-383d-4728-9336-ff558cd83ad1,3d663a99-383d-4728-9336-ff558cd83ad1,,"Noyes, Pamela D.; McElwee, Matthew K.; Miller, Hilary D.; Clark, Bryan W.; Van Tiem, Lindsey A.; Walcott, Kia C.; Erwin, Kyle N.; Levin, Edward D.",10.1016/j.envint.2009.02.006,,0160-4120,6,"Environment International",,,"Ch5,7,8,9",971-986,"The toxicology of climate change: Environmental contaminants in a warming world",35,2009,0,16485,3d663a99-383d-4728-9336-ff558cd83ad1,"Journal Article",/article/10.1016/j.envint.2009.02.006
/reference/3d74b037-dfba-424c-a08d-819f7d3f6a4d,https://data.globalchange.gov/reference/3d74b037-dfba-424c-a08d-819f7d3f6a4d,3d74b037-dfba-424c-a08d-819f7d3f6a4d,,"Roelofs, Cora; Wegman, David",10.2105/AJPH.2014.302145,,1541-0048,10,"American Journal of Public Health",,,,1799-1801,"Workers: The climate canaries",104,2014,0,19259,3d74b037-dfba-424c-a08d-819f7d3f6a4d,"Journal Article",/article/10.2105/AJPH.2014.302145
/reference/3d995371-0fe3-44b3-9786-92d909982b46,https://data.globalchange.gov/reference/3d995371-0fe3-44b3-9786-92d909982b46,3d995371-0fe3-44b3-9786-92d909982b46,,"Halsby, K.D.; Joseph, C.A.; Lee, J.V.; Wilkinson, P.",10.1017/S0950268813003294,,1469-4409,11,"Epidemiology & Infection",,,,2352-2359,"The relationship between meteorological variables and sporadic cases of Legionnaires' disease in residents of England and Wales",142,2014,0,19156,3d995371-0fe3-44b3-9786-92d909982b46,"Journal Article",/article/10.1017/S0950268813003294
/reference/3dca835b-beb7-49f5-b9c1-54753dc54ddc,https://data.globalchange.gov/reference/3dca835b-beb7-49f5-b9c1-54753dc54ddc,3dca835b-beb7-49f5-b9c1-54753dc54ddc,"Rationale: Certain outdoor air pollutants cause asthma exacerbations in children. To advance understanding of these relationships, further characterization of the dose–response and pollutant lag effects are needed, as are investigations of pollutant species beyond the commonly measured criteria pollutants. Objectives: Investigate short-term associations between ambient air pollutant concentrations and emergency department visits for pediatric asthma. Methods: Daily counts of emergency department visits for asthma or wheeze among children aged 5 to 17 years were collected from 41 Metropolitan Atlanta hospitals during 1993–2004 (n = 91,386 visits). Ambient concentrations of gaseous pollutants and speciated particulate matter were available from stationary monitors during this time period. Rate ratios for the warm season (May to October) and cold season (November to April) were estimated using Poisson generalized linear models in the framework of a case-crossover analysis. Measurements and Main Results: Both ozone and primary pollutants from traffic sources were associated with emergency department visits for asthma or wheeze; evidence for independent effects of ozone and primary pollutants from traffic sources were observed in multipollutant models. These associations tended to be of the highest magnitude for concentrations on the day of the emergency department visit and were present at relatively low ambient concentrations. Conclusions: Even at relatively low ambient concentrations, ozone and primary pollutants from traffic sources independently contributed to the burden of emergency department visits for pediatric asthma.","Strickland, Matthew J.; Darrow, Lyndsey A.; Klein, Mitchel; Flanders, W. Dana; Sarnat, Jeremy A.; Waller, Lance A.; Sarnat, Stefanie E.; Mulholland, James A.; Tolbert, Paige E.",10.1164/rccm.200908-1201OC,"04/08 08/06/received 03/31/accepted",1535-4970,3,"American Journal of Respiratory and Critical Care Medicine",,,"ajrccm1823307[PII] 20378732[pmid] Am J Respir Crit Care Med",307-316,"Short-term associations between ambient air pollutants and pediatric asthma emergency department visits",182,2010,0,19097,3dca835b-beb7-49f5-b9c1-54753dc54ddc,"Journal Article",/article/10.1164/rccm.200908-1201OC
/reference/3dfa7576-6fcc-4db8-8d1a-cc81c6cba6cf,https://data.globalchange.gov/reference/3dfa7576-6fcc-4db8-8d1a-cc81c6cba6cf,3dfa7576-6fcc-4db8-8d1a-cc81c6cba6cf,"The ability of the invading NY99 strain of West Nile virus (WNV) to elicit an elevated viremia response in California passerine birds was critical for the effective infection of Culex mosquitoes. Of the bird species tested, Western scrub jays, Aphelocoma coerulescens, produced the highest viremia response, followed by house finches, Carpodacus mexicanus, and house sparrows, Passer domesticus. Most likely, few mourning, Zenaidura macroura, or common ground, Columbina passerine, doves and no California quail, Callipepla californica, or chickens would infect blood-feeding Culex mosquitoes. All Western scrub jays and most house finches succumbed to infection. All avian hosts produced a lower viremia response and survived after infection with an endemic strain of St. Louis encephalitis virus. Culex species varied in their susceptibility to infection with both viruses, with Culex stigmatosoma Dyar generally most susceptible, followed by Culex tarsalis Coquillett, and then Culex p. quinquefasciatus Say. Populations within Culex species varied markedly in their susceptibility, perhaps contributing to the focality of WNV amplification. Transmitting female Cx. tarsalis expectorated from six to 3,777 plaque-forming units (PFU) of WNV during transmission trials, thereby exposing avian hosts to a wide range of infectious doses. Highly susceptible house finches and moderately susceptible mourning doves were infected by subcutaneous inoculation with decreasing concentrations of WNV ranging from 15,800 to <0.3 PFU. All birds became infected and produced comparable peak viremias on days 2-3 postinoculation; however, the rise in viremia titer and onset of the acute phase of infection occurred earliest in birds inoculated with the highest doses. WNV virulence in birds seemed critical in establishing elevated viremias necessary to efficiently infect blood feeding Culex mosquitoes.","Reisen, W. K.; Fang, Y.; Martinez, V. M.",10.1603/0022-2585(2005)042[0367:ahamdc]2.0.co;2,May,1938-2928,3,"Journal of Medical Entomology","Animals; Bird Diseases/virology; Birds/*virology; Chickens/virology; Culex/*virology; Disease Susceptibility/veterinary; Encephalitis Virus, St. Louis; Encephalitis, St. Louis/*transmission; Female; Insect Vectors/*virology; Passeriformes/virology; Species Specificity; Viremia/veterinary; West Nile Fever/*transmission; West Nile virus",,"Reisen, W K Fang, Y Martinez, V M eng R01-A155607/PHS HHS/ R01-AI39483/AI/NIAID NIH HHS/ Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, P.H.S. 2005/06/21 09:00 J Med Entomol. 2005 May;42(3):367-75.",367-375,"Avian host and mosquito (Diptera: Culicidae) vector competence determine the efficiency of West Nile and St. Louis encephalitis virus transmission",42,2005,0,18028,3dfa7576-6fcc-4db8-8d1a-cc81c6cba6cf,"Journal Article",/article/10.1603/0022-2585(2005)042%5B0367:ahamdc%5D2.0.co;2
/reference/3dfe4793-add4-44b8-b6d4-6aecd0561ede,https://data.globalchange.gov/reference/3dfe4793-add4-44b8-b6d4-6aecd0561ede,3dfe4793-add4-44b8-b6d4-6aecd0561ede,,"Berdalet, Elisa; Peters, Francesc; Koumandou, V. Lila; Roldán, Cristina; Guadayol, Òscar; Estrada, Marta",10.1111/j.1529-8817.2007.00392.x,,1529-8817,5,"Journal of Phycology",,,,965-977,"Species-specific physiological response of dinoflagellates to quantified small-scale turbulence",43,2007,0,16654,3dfe4793-add4-44b8-b6d4-6aecd0561ede,"Journal Article",/article/10.1111/j.1529-8817.2007.00392.x
/reference/3e34582d-ee92-45b3-9240-924ca5e98824,https://data.globalchange.gov/reference/3e34582d-ee92-45b3-9240-924ca5e98824,3e34582d-ee92-45b3-9240-924ca5e98824,,"Corsi, S. R.; Borchardt, M. A.; Spencer, S. K.; Hughes, P. E.; Baldwin, A. K.",10.1016/j.scitotenv.2014.05.072,,1879-1026,,"Science of The Total Environment",,,,849-860,"Human and bovine viruses in the Milwaukee River watershed: Hydrologically relevant representation and relations with environmental variables",490,2014,0,18859,3e34582d-ee92-45b3-9240-924ca5e98824,"Journal Article",/article/10.1016/j.scitotenv.2014.05.072
/reference/3e64a90d-f0c2-4f1a-a286-ce437bd95e60,https://data.globalchange.gov/reference/3e64a90d-f0c2-4f1a-a286-ce437bd95e60,3e64a90d-f0c2-4f1a-a286-ce437bd95e60,,"Salvadori, Marina I.; Sontrop, Jessica M.; Garg, Amit X.; Moist, Louise M.; Suri, Rita S.; Clark, William F.",10.1038/ki.2008.616,,1523-1755,,"Kidney International",,,,S33-S34,"Factors that led to the Walkerton tragedy",75,2009,0,16169,3e64a90d-f0c2-4f1a-a286-ce437bd95e60,"Journal Article",/article/10.1038/ki.2008.616
/reference/3ee02b96-55f8-4582-8d49-6865f6f0a603,https://data.globalchange.gov/reference/3ee02b96-55f8-4582-8d49-6865f6f0a603,3ee02b96-55f8-4582-8d49-6865f6f0a603,"Exposure to adverse life events typically predicts subsequent negative effects on mental health and well-being, such that more adversity predicts worse outcomes. However, adverse experiences may also foster subsequent resilience, with resulting advantages for mental health and well-being. In a multiyear longitudinal study of a national sample, people with a history of some lifetime adversity reported better mental health and well-being outcomes than not only people with a high history of adversity but also than people with no history of adversity. Specifically, U-shaped quadratic relationships indicated that a history of some but nonzero lifetime adversity predicted relatively lower global distress, lower self-rated functional impairment, fewer posttraumatic stress symptoms, and higher life satisfaction over time. Furthermore, people with some prior lifetime adversity were the least affected by recent adverse events. These results suggest that, in moderation, whatever does not kill us may indeed make us stronger.","Seery, M. D.; Holman, E. A.; Silver, R. C.",10.1037/a0021344,Dec,1939-1315,6,"Journal of Personality and Social Psychology","*Adaptation, Psychological; Adolescent; Adult; Aged; Aged, 80 and over; Female; Humans; *Life Change Events; Likelihood Functions; Longitudinal Studies; Male; Middle Aged; Models, Psychological; *Resilience, Psychological; Stress, Psychological/*psychology; United States",eng,"1939-1315 Seery, Mark D Holman, E Alison Silver, Roxane Cohen T32 MH19958/MH/NIMH NIH HHS/United States Comparative Study Journal Article Research Support, N.I.H., Extramural Research Support, U.S. Gov't, Non-P.H.S. United States J Pers Soc Psychol. 2010 Dec;99(6):1025-41. doi: 10.1037/a0021344.",1025-1041,"Whatever does not kill us: Cumulative lifetime adversity, vulnerability, and resilience",99,2010,0,18179,3ee02b96-55f8-4582-8d49-6865f6f0a603,"Journal Article",/article/10.1037/a0021344
/reference/3f2402c5-22aa-4f75-861e-f6aca127cd1f,https://data.globalchange.gov/reference/3f2402c5-22aa-4f75-861e-f6aca127cd1f,3f2402c5-22aa-4f75-861e-f6aca127cd1f,,"Frumkin, H.
Hess, J.
Luber, G.
Malilay, J.
McGeehin, M.",10.2105/AJPH.2007.119362,,0090-0036,3,"American Journal of Public Health",,,,435-445,"Climate change: The public health response",98,2008,0,352,3f2402c5-22aa-4f75-861e-f6aca127cd1f,"Journal Article",/article/10.2105/AJPH.2007.119362